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Case Report
Methemoglobinemia development after ingestion of a chinese herbal medicine: A case report
Se Eun Hyun, Pil Joo Hwang, Kyung Pil Nam, Eun Gyong Yoo, Man Yong Han
Clin Exp Pediatr. 2008;52(3):385-388.   Published online March 15, 2009
An 8-year-old girl visited the emergency room with perioral cyanosis and dyspnea, which had developed 20 hrs prior to the hospital visit. She had taken a Chinese herbal medication for 3 days prior to the onset of the symptoms. A co-oximeter examination revealed a methemoglobin level of 23.7%. An intravenous infusion of methylene blue was administered. Chemical analysis of the...
Original Article
Methemoglobinemia Associated with Diarrheal Disease before 2 Months of Ag
Ji Ah Jung, Jung Won Lee, Jeong Wan Seo
Clin Exp Pediatr. 2001;44(12):1387-1394.   Published online December 15, 2001
Purpose : Methemoglobinemia has been observed during a diarrheal illness among those who were not exposed to oxidizing agents in neonates and young infants. We performed the study to determine predisposing factors in this entity. Methods : The medical records of 24 patients were reviewed, who were admitted for diarrhea with methemoglobinemia to Ewha Womans University MokDong Hospital, from February 1994 to November 2000. The...
Case Report
A Case of Congenital Methemoglobinemia Due to NADH-Methemoglobin Reductase Deficiency
Do Hyeon Kim, Jin A Lee, Sung Jae Lee, Heui Seung Jo, Yun Kyoung Lee, June Dong Park, Beyong Il Kim, Jung-Hwan Choi, Dong Soon Lee, Han Ik Cho, Kyung Ran Park
Clin Exp Pediatr. 2001;44(6):699-704.   Published online June 15, 2001
Congenital methemoglobinemia is caused by NADH-methemoglobin reductase deficiency in more than half of the total reported cases. NADH-methemoglobin reductase deficiency is an uncommon hereditary disorder producing methemoglobinemia and cyanosis in the homozygous subject. A majority of the patients born with these abnormalities have only a cosmetic defect-asymptomatic cyanosis. Congenital methemoglobinemia due to NADH-methemoglobin reductase deficiency is an autosomal recessive disorder...
Original Article
Changes in Incidence of Infantile Transient Methemoglobinemia Associated with Infectious Diarrhea during 1989-1999 in Kyungnam Province
Young Suk Kim, Eun-A Kim, Jae-Young Lim, Myoung-Bum Choi, Chan-Hoo Park, Hyang-Ok Woo, Byung-Kiu Park, Hee-Shang Youn, Yang-Suk Jung, Dong-Jin Lee, Jung Hee Kim, Eun Jeong Kim, Chul Kyu Kim
Clin Exp Pediatr. 2000;43(12):1569-1576.   Published online December 15, 2000
Purpose : Infantile transient methemoglobinemia(ITM) may develop in association with infectious diarrhea without exposure to any toxic oxidizing agents. We observed that the number of ITM associated with infectious diarrhea have increased at the Gyeongsang National University Hospital (GNUH), located in the western area of Kyungnam province during the last 4 years. To determine whether this phenomenon was similarily observed...
Analysis of Contaminated Ground Water Inducing Methemoglobinemia and Epidemiologic Investigation of Contaminated Ground Water
Bu Heon Lee, Ji Young Suh, Nam Su Kim, Hang Lee, Young Yeul Kim, Bo Youl Choi, Hung Bae Park, Min Young Kim, In Hak Yeo
Clin Exp Pediatr. 1995;38(4):507-512.   Published online April 15, 1995
Purpose : Drining of Nitrate-contaminated water has been the most common cause of acquired methemoglobinemia. We had cyanotic infant diagnosed as methemoglobinemia caused by feeding with powdered formula mixed with nitrate-contaminated ground water. Methods : We had done epidemiologic investigation to identify the cause of an infant's methemoglobinemia. Analysis of ground water and blood test of involved family members and neighbors...
Case Report
Transient Methemoglobinemia with Acidosis in Newborn
Heng Mi Kim
Clin Exp Pediatr. 1994;37(3):416-421.   Published online March 15, 1994
methemoglobinemia is an uncommon clinicl problem generally caused by inherited disorders of hemoglobin metabolism or environmental toxicity from oxidizing agents. Since methemoglobin has no oxigen carrying capacity, patient with severe methemoglobinemia may have dangerous hypoxia even when arterial oxygen tension is normal. Degree of exposure to oxidants which are benign for older individuals may produce severe methemoglobinemia in newborns. Acidosis without...
Original Article
Clinical Observation of Dds Induced Methemoglobinemia.
Hee Seon Auh, Kwan Hwooy Cho, Kir Young Kim, Duk Jin Yun, Sook Pyo Kwon
Clin Exp Pediatr. 1983;26(3):257-264.   Published online March 31, 1983
Various compounds activate the oxidation of hemoglobin. These include nitrites, sulfonamides, aniline derivatives, chlorates, quinones, acetanilid and phenacetin. The striking offender has been DDS in infancy and childhood who live in Kangwon Do, Korea. The common untoward effects of DDS are hemolytic anemia and methemoglobinemia. This clinical study was carried out for analysis on 57 cases which were diagnosed as DDS induced methemoglobinemia at...
Clinical Observation of The Methemoglobinemia in Childrens.
Hak Hew Shin, Chull Kwon Chung, Myung Hi Shin, Ji Sub Oh
Clin Exp Pediatr. 1982;25(11):1131-1140.   Published online November 30, 1982
The clinical records of 28 cases with. Methemoglobinemia who were admitted to the Dept, of pediatrics, WMBH Busan from January 1978 to December 1981 were reviewed. The following results were obtained. 1) Incidence of Methemoglobinemia, was 17.8% among the all drug intoxication and seasonal incidence was peak in spring(50%). 2) The incidence was higher in boys than girls(60.7%), the highest incidence was noted in the age of 1...
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